Collaboration with APIS Assay Technologies to assess the suitability of TROLL-2 and TROLL-3 as predictive biomarkers of cancer progression and their role as markers of chemotherapy response


 Pan-cancer analysis reveals TAp63-regulated oncogenic lncRNAs that promote cancer progression through AKT activation


TAp63-Regulated miRNAs Suppress Cutaneous Squamous Cell Carcinoma through Inhibition of a Network of Cell-Cycle Genes


∆Np63/DGCR8- Dependent MicroRNAs Mediate Therapeutic Efficacy of HDAC Inhibitors in Cancer.


IAPP-driven metabolic reprogramming induces regression of p53-deficient tumours in vivo. 

TAp63 is the master transcriptional regulator of lipid and glucose metabolism 

TAp63 suppresses metastasis through coordinate regulation of Dicer and miRNAs


DeltaNp63 transcriptionally regulates brachyury, a gene with diverse roles in limb development, tumorigenesis and metastasis

TAp63 prevents premature aging by promoting adult stem cell maintenance


p63 and p73 are required for p53-dependent apoptosis in response to DNA damage

  • Urist M. and Prives C. p53 leans on its siblings. Cancer Cell. 2002 May 01. doi: 10.1016/S1535-6108(02)00064-8.
  • Mitchell A. Apoptosis: The p53 mafia. Nature Reviews Cancer. 2002 May 01. doi: 10.1038/nrc804.
  • Faculty of 1000 – rated 6 (William Kaelin, M.D., Professor, Harvard Medical School, Dana Farber Cancer Institute)